NS FL 9 BB Q32

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mgarc805
Posts: 35
Joined: Tue Jun 25, 2019 11:23 am

NS FL 9 BB Q32

Post by mgarc805 » Thu Aug 08, 2019 2:41 pm

I know that in the MCAT one passage is not supposed to influence the questions of the other passage but I also know that the MCAT offers true information (they don't make up problems or process that don't exits). In this RN question I got confused because in the passage right after this one (passage 7) the first paragraph says that T2DM has the opposite problem of too much insulin production.
NS_Tutor_Mathias
Posts: 264
Joined: Sat Mar 30, 2019 8:39 pm

Re: NS FL 9 BB Q32

Post by NS_Tutor_Mathias » Thu Aug 08, 2019 4:28 pm

Both are actually correct - the subsequent passage describes the pathophysiology of T2DM well, but does not explicitly mention that B-islets tend to fail in late-stage T2DM.


This is simply a discrete bit of knowledge being tested, fairly unrelated to the passage itself: The common progression of T2DM. I understand this is potentially frustrating, but this sort of breadth of background knowledge is part of the MCAT. Luckily, clinically-adjacent details like this you are only responsible for on diseases mentioned in the AAMC content outline.

If you'd like to know a bit more about diabetes, this is a good analysis of your exact question:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4435650/
mgarc805
Posts: 35
Joined: Tue Jun 25, 2019 11:23 am

Re: NS FL 9 BB Q32

Post by mgarc805 » Mon Aug 12, 2019 12:00 pm

Ok, So based on the practice test and the article you sent me, the pathophysiology of T2D is that close to the onset of T2D Beta cells have an overproduction of insulin that's product of elevated blood sugar due to unresponsive cells in the liver muscle and adipose tissue but later on the disease this production leads to malfunction of the beta cells.

Thank you
NS_Tutor_Mathias
Posts: 264
Joined: Sat Mar 30, 2019 8:39 pm

Re: NS FL 9 BB Q32

Post by NS_Tutor_Mathias » Wed Aug 14, 2019 4:56 pm

Aye
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